Search results for "delayed effects"

showing 10 items of 138 documents

Dietary and Household Sources of Prenatal Exposure to Polybrominated Diphenyl Ethers (PBDEs) in the INMA Birth Cohort (Spain)

2016

This study looked at predictors of exposure to polybrominated diphenyl ethers (PBDEs) with a focus on dietary and household-level factors. Concentrations of BDE-47, -99, -153, and -209 and their sum (∑PBDEs) were measured in cord serum. Spanish women (n = 541) completed two semiquantitative food frequency questionnaires during the first and third trimesters of pregnancy. The daily mean intake (grams) of eggs, dairy products, meat, cereals and pasta, vegetables and pulses, fruits, shellfish and cephalopods, and fish, and the weekly mean intake (servings) of lean, large oily, other oily, and other fish from both questionnaires were averaged. Information on house size, curtains and carpets at …

010501 environmental sciencesThird trimester01 natural sciences03 medical and health sciences0302 clinical medicinePolybrominated diphenyl ethersPregnancyCORD SERUMEnvironmental healthHalogenated Diphenyl EthersAnimalsHumansEnvironmental ChemistryMedicine030212 general & internal medicinePrenatal exposure0105 earth and related environmental sciencesPregnancyFood frequencybusiness.industryGeneral Chemistrymedicine.diseaseDiet3. Good healthSeafoodSpainPrenatal Exposure Delayed EffectsEnvironmental chemistryGestationFemalebusinessBirth cohortEnvironmental Science & Technology
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Topical bioassay of Oberon® on Drosophila melanogaster pupae: delayed effects on ovarian proteins, cuticular hydrocarbons and sexual behaviour

2021

International audience; Spiromesifen (Oberon? 240 SC; concentrated solution; Bayer CropScience, Germany) a pesticide derived from spirocyclic tetronic acids, acts mainly though blocking lipid biosynthesis via acetyl-CoA carboxylase. However, its mode of action requires further analyses. This study aims to evaluate the sublethal effects of spiromesifen on the fruit fly Drosophila melanogaster. The treatment was carried out by topical application on newly moulted pupae using two concentrations (LD10 = 21.45 and LD25 = 39.53 ?g a.i./pupa). Our results showed that the developmental duration of larvae and pupae were not significantly affected by treatment. However, spiromesifen caused delayed ef…

0106 biological sciencesanimal structuresprogeny010603 evolutionary biology01 natural sciencesOberonLipid biosynthesisBioassaycomputer.programming_languagebiology010604 marine biology & hydrobiologycuticular hydrocarbonsfungisexual behaviour[SDV.BDLR]Life Sciences [q-bio]/Reproductive BiologyPesticidebiology.organism_classificationspiromesifenproteinsPupaBiochemistryAnimal Science and ZoologyhexapodaDrosophila melanogasterdelayed effectscomputerDevelopmental Biology
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Alterations in Tau Protein Level and Phosphorylation State in the Brain of the Autistic-Like Rats Induced by Prenatal Exposure to Valproic Acid

2021

Autism spectrum disorder (ASD) is a neurodevelopmental condition characterized by deficient social interaction and communication besides repetitive, stereotyped behaviours. A characteristic feature of ASD is altered dendritic spine density and morphology associated with synaptic plasticity disturbances. Since microtubules (MTs) regulate dendritic spine morphology and play an important role in spine development and plasticity the aim of the present study was to investigate the alterations in the content of neuronal α/β-tubulin and Tau protein level as well as phosphorylation state in the valproic acid (VPA)-induced rat model of autism. Our results indicated that maternal exposure to VPA indu…

0301 basic medicineDendritic spineHippocampuslcsh:Chemistry0302 clinical medicinePregnancyTubulinPhosphorylationlcsh:QH301-705.5SpectroscopyValproic AcidbiologyERK1/2Chemistryautism spectrum disorders (ASD)valproic acid (VPA)BrainGeneral MedicineImmunohistochemistryComputer Science Applicationsmedicine.anatomical_structureCerebral cortexMaternal ExposurePrenatal Exposure Delayed EffectsFemaleDisease Susceptibilitymedicine.drugSignal Transductionmedicine.medical_specialtyCDK5Tau proteintau ProteinsCatalysisArticleInorganic Chemistry03 medical and health sciencesInternal medicinemental disordersmedicineAnimalsPhysical and Theoretical ChemistryAutistic DisorderMolecular BiologyCyclin-dependent kinase 5GSK-3βValproic AcidOrganic Chemistryα/β-tubulinRatsEnzyme Activation030104 developmental biologyEndocrinologylcsh:Biology (General)lcsh:QD1-999MAP-TauChromatolysisSynaptic plasticitybiology.proteinAkt/mTOR signalling030217 neurology & neurosurgeryBiomarkersInternational Journal of Molecular Sciences
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Mothering under the influence: How perinatal drugs of abuse alter the mother-infant interaction

2018

AbstractAlthough drug-abusing women try to moderate their drug and alcohol use during pregnancy, they often relapse at a time when childcare needs are high and maternal bonding is critical to an infant’s development. In the clinical setting, the search for the neural basis of drug-induced caregiving deficits is complex due to several intervening variables. Rather, the preclinical studies that control for drug dose and regimen, as well as for gestational and postpartum environment, allow a precise determination of the effects of drugs on maternal behaviour. Given the relevance of the issue, this review will gather reports on the phenotypic correlates of maternal behaviour in preclinical stud…

0301 basic medicineDrugMalecannabiSubstance-Related Disordersmedia_common.quotation_subjectMother infantcocainePrenatal Exposure Delayed EffectMaternal behaviourNicotine03 medical and health sciences0302 clinical medicinePregnancymedicineHumansMaternal Behaviormedia_commonPregnancymaternal behaviourbiologybusiness.industryalcoholGeneral Neurosciencestimulantperinatal substance usemedicine.diseasebiology.organism_classificationSubstance-Related DisorderMother-Child RelationsopiateRegimen030104 developmental biologyPrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaGestationFemaleCannabisbusiness030217 neurology & neurosurgeryClinical psychologymedicine.drugnicotineHuman
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Prenatal exposure to mixtures of xenoestrogens and genome-wide DNA methylation in human placenta

2015

BACKGROUND: In utero exposure to xenostrogens may modify the epigenome. We explored the association of prenatal exposure to mixtures of xenoestrogens and genome-wide placental DNA methylation. MATERIALS & METHODS: Sex-specific associations between methylation changes in placental DNA by doubling the concentration of TEXB-alpha exposure were evaluated by robust multiple linear regression. Two CpG sites were selected for validation and replication in additional male born placentas. RESULTS: No significant associations were found, although the top significant CpGs in boys were located in the LRPAP1, HAGH, PPARGC1B, KCNQ1 and KCNQ1DN genes, previously associated to birth weight, Type 2 diabetes…

0301 basic medicineMaleCancer Researchmedicine.medical_specialtymedicine.medical_treatmentPlacentaADNBiologyEpigenesis GeneticAndrology03 medical and health scienceschemistry.chemical_compoundSex FactorsPregnancyPlacentaInternal medicineGeneticsmedicineBirth WeightHumansPrenatalLDL-Receptor Related Protein-Associated ProteinGeneTEXBEndocrine disruptorsDNA methylationEpigenome xenoestrogensRNA-Binding ProteinsEstrogensMethylationEpigenomeSteroid hormone030104 developmental biologyEndocrinologymedicine.anatomical_structureXenoestrogenCpG sitechemistryPrenatal Exposure Delayed EffectsDNA methylationKCNQ1 Potassium ChannelProgrammingCpG IslandsFemaleThiolester HydrolasesCarrier ProteinsGenome-Wide Association Study
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Maternal inflammation has a profound effect on cortical interneuron development in a stage and subtype-specific manner

2018

AbstractSevere infections during pregnancy are one of the major risk factors for cognitive impairment in the offspring. It has been suggested that maternal inflammation leads to dysfunction of cortical GABAergic interneurons that in turn underlies cognitive impairment of the affected offspring. However, the evidence comes largely from studies of adult or mature brains and how the impairment of inhibitory circuits arises upon maternal inflammation is unknown. Here we show that maternal inflammation affects multiple steps of cortical GABAergic interneuron development, i.e., proliferation of precursor cells, migration and positioning of neuroblasts, as well as neuronal maturation. Importantly,…

0301 basic medicineMaleInterneuronOffspringNeurogenesisMothersInflammationBiologyInhibitory postsynaptic potentialArticle03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineNeuroblastCell MovementInterneuronsPregnancyPrecursor cellmedicineAnimalsCognitive DysfunctionGABAergic NeuronsMolecular BiologyCell ProliferationCerebral CortexInflammationPregnancyCell growthNeurogenesisCognitionmedicine.diseaseMice Inbred C57BLPsychiatry and Mental health030104 developmental biologymedicine.anatomical_structurenervous systemPrenatal Exposure Delayed EffectsGABAergicFemalemedicine.symptomPsychiatric disordersNeuroscience030217 neurology & neurosurgeryNeuroscience
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Prenatal exposure to endocrine disrupting chemicals and risk of being born small for gestational age: Pooled analysis of seven European birth cohorts

2018

Background and aims: There is evidence that endocrine disrupting chemicals (EDCs) have developmental effects at environmental concentrations. We investigated whether some EDCs are associated with the adverse birth outcome Small for Gestational Age (SGA). Methods: We used PCB 153, p,p'-DDE, HCB, PFOS and PFOA measured in maternal, cord blood or breast milk samples of 5446 mother-child pairs (subset of 693 for the perfluorinated compounds) from seven European birth cohorts (1997–2012). SGA infants were those with birth weight below the 10th percentile for the norms defined by gestational age, country and infant's sex. We modelled the association between measured or estimated cord serum EDC co…

0301 basic medicineMalemedicine.medical_specialtyBirth weight010501 environmental sciencesBreast milkEndocrine Disruptors01 natural sciencesPooled analysis03 medical and health sciencesSDG 3 - Good Health and Well-beingInterquartile rangePregnancyMedicine[SDV.EE.SANT] Life Sciences [q-bio]/Ecology environment/HealthHumansBiologylcsh:Environmental sciences0105 earth and related environmental sciencesGeneral Environmental Science2. Zero hungerlcsh:GE1-350[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthPregnancyMilk Humanbusiness.industryObstetricsSmokingInfant NewbornGestational ageEnvironmental exposureOdds ratioEndocrine disrupting chemicals (EDCs)Infant Low Birth Weightmedicine.diseaseFetal Blood3. Good healthChemistrySmall for gestational age (SGA)030104 developmental biology[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieMaternal ExposurePrenatal Exposure Delayed EffectsSmall for gestational age/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemale[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologiebusiness
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DNA methylation links prenatal smoking exposure to later life health outcomes in offspring

2019

Background Maternal smoking during pregnancy is associated with adverse offspring health outcomes across their life course. We hypothesize that DNA methylation is a potential mediator of this relationship. Methods We examined the association of prenatal maternal smoking with offspring blood DNA methylation in 2821 individuals (age 16 to 48 years) from five prospective birth cohort studies and perform Mendelian randomization and mediation analyses to assess whether methylation markers have causal effects on disease outcomes in the offspring. Results We identify 69 differentially methylated CpGs in 36 genomic regions (P value < 1 × 10−7) associated with exposure to maternal smoking in adolesc…

0301 basic medicinePhysiologyraskausDiseaseBioinformaticsEpigenesis Genetic/dk/atira/pure/core/keywords/icepCohort Studies0302 clinical medicinePregnancyGTP-Binding Protein gamma SubunitsEpidemiologySCHIZOPHRENIADiseaseLongitudinal StudiesProspective StudieskohorttitutkimusGenetics (clinical)Maternal smokingGenetics & HeredityRISK0303 health sciencesDNA methylationSmokingWIDEMethylationASSOCIATIONMiddle AgedDNA-metylaatio3. Good healthCausalityPREGNANCYOncologyMaternal ExposureSchizophreniaPrenatal Exposure Delayed Effects030220 oncology & carcinogenesisDNA methylationkausaliteettilifecourseLife course approachFemaleICEPLife Sciences & BiomedicineAdultTOBACCO-SMOKEMediation (statistics)medicine.medical_specialtyAdolescentOffspringBirth weightPersistenceYoung Adult03 medical and health sciencestupakointiterveysvaikutuksetMendelian randomizationGeneticsmedicineHumansMolecular BiologyMETAANALYSIS030304 developmental biologyPregnancyScience & TechnologyIDENTIFICATIONbusiness.industryMATERNAL CIGARETTE-SMOKINGResearchMediationLife courseMendelian Randomization Analysismedicine.diseaseBIRTH-WEIGHT030104 developmental biologyCpG Islandsbusiness030217 neurology & neurosurgeryGenome-Wide Association StudyDevelopmental Biology
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Gynecologic cancers in pregnancy: guidelines based on a third international consensus meeting

2019

We aimed to provide comprehensive protocols and promote effective management of pregnant women with gynecological cancers. New insights and more experience have been gained since the previous guidelines were published in 2014. Members of the International Network on Cancer, Infertility and Pregnancy (INCIP), in collaboration with other international experts, reviewed existing literature on their respective areas of expertise. Summaries were subsequently merged into a manuscript that served as a basis for discussion during the consensus meeting. Treatment of gynecological cancers during pregnancy is attainable if management is achieved by collaboration of a multidisciplinary team of health c…

0301 basic medicinemedicine.medical_specialtyCognitiveGenital Neoplasms FemaleInternational Cooperation[SDV.CAN]Life Sciences [q-bio]/CancerCancer Chemotherapy Cognitive Gynecologic Offspring Pregnancy[SDV.MHEP.GEO]Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetrics03 medical and health sciencesGenital Neoplasms Female/therapyGynecologicOffspring0302 clinical medicineSDG 3 - Good Health and Well-beingPregnancyHealth caremedicinePrenatal Exposure Delayed Effects/etiologyHumansChemotherapyRadiation treatment planningIntensive care medicineSocieties MedicalCancerPregnancy Complications Neoplastic/therapyPregnancyVaginal deliverybusiness.industryCancerHematologymedicine.diseasePrognosis3. Good healthCancer registryLong-term care030104 developmental biologyOncology030220 oncology & carcinogenesisPrenatal Exposure Delayed EffectsPractice Guidelines as TopicFemalePractice Guidelines as Topic/standardsbusinessBreast feedingPregnancy Complications Neoplastic
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Potential risks to offspring of intrauterine exposure to maternal age-related obstetric complications

2016

Several hypotheses have been proposed to explain the negative effects of delayed motherhood on an offspring’s morbidity later in life. However, these hypotheses are not supported by clinical and epidemiological evidence. Because advanced maternal age is associated with increased risk of obstetric complications, the aim of the present study was to ascertain whether the negative effects on offspring of intrauterine exposure to maternal age-related obstetric complications may explain the reported negative effects of delayed motherhood on offspring. To this end, a literature search was performed to identify relevant publications up to March 2016 on PubMed; references cited in relevant articles …

0301 basic medicinemedicine.medical_specialtyOffspringmedia_common.quotation_subjectFertilityReproductive technologyBiologyMaternal PhysiologyEpigenesis Genetic03 medical and health sciences0302 clinical medicineEndocrinologyPregnancyRisk FactorsGeneticsmedicineHumansFertility preservationAdvanced maternal ageMolecular Biologymedia_commonPregnancy030219 obstetrics & reproductive medicineMaternal TransmissionObstetricsAge FactorsPregnancy Outcomemedicine.diseasePregnancy Complications030104 developmental biologyReproductive MedicinePrenatal Exposure Delayed EffectsFemaleAnimal Science and ZoologyMaternal AgeDevelopmental BiologyBiotechnologyReproduction, Fertility and Development
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